Patients that suffer from hyperinsulinemia may have a decreased number of insulin receptors on their cell membranes. This is an example of which phenomenon?

A) Down-regulation
B) Hyposecretion
C) Permissiveness
D) Receptor inactivation

Answers

Answer 1

I think the right answer is hyposecetion which is b


Related Questions

Ethanol (CH3CH2OH) is an alcohol found in beverages. It is oxidized in the body to acetaldehyde by the enzyme alcohol dehydrogenase. Methanol (CH3OH), also known as wood alcohol, is converted to formaldehyde by the same enzyme. Acetaldehyde is toxic, but formaldehyde is far more toxic to humans, which is why the ingestion of relatively small amounts of methanol can cause blindness or death. One treatment for mild methanol poisoning is the administration of ethanol. Why might a doctor choose this treatment? Ethanol (CH3CH2OH) is an alcohol found in beverages. It is oxidized in the body to acetaldehyde by the enzyme alcohol dehydrogenase. Methanol (CH3OH), also known as wood alcohol, is converted to formaldehyde by the same enzyme. Acetaldehyde is toxic, but formaldehyde is far more toxic to humans, which is why the ingestion of relatively small amounts of methanol can cause blindness or death. One treatment for mild methanol poisoning is the administration of ethanol. Why might a doctor choose this treatment?

Answers

A doctor might choose to administer ethanol since ethanol has a greater affinity for alcohol dehydrogenase than methanol, thus reducing the formation of the toxic metabolites of methanol. Excess methanol is then removed from the body by hemodialysis

Final answer:

A doctor might choose to administer ethanol to treat mild methanol poisoning because it competes with methanol for the enzyme alcohol dehydrogenase, reducing the toxic effects of formaldehyde. Ethanol acts as a competitive inhibitor, blocking the conversion of methanol to formaldehyde.

Explanation:

A doctor might choose to administer ethanol as a treatment for mild methanol poisoning because ethanol competes with methanol for the enzyme alcohol dehydrogenase. By administering ethanol, the amount of methanol that is converted to formaldehyde is reduced, minimizing the toxic effects of formaldehyde on the body. Ethanol serves as a competitive inhibitor, effectively blocking the conversion of methanol to formaldehyde and allowing the body more time to naturally eliminate the methanol.

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